GE Healthcare Arterial Pressure Variation Quick Guide

GE Healthcare
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Quick Guide
What is arterial pressure variation?
Positive pressure ventilation causes blood pressure
changes in the chest cavity: inspiratory phase increases
the pressure and expiratory phase decreases it. The magnitude of these changes is dependent on the uid status of the patient. In hypovolemic patients these swings are higher in amplitude compared to normovolemic or hypervolemic patients. This is a well known phenomenon.
The arterial pressure variation algorithm looks for respiratory changes in the arterial blood pressure. It only uses the arterial blood pressure analysis and does not use any other parameter to recognize the
respiratory cycle.
Arterial pressure variation can be used to assess uid responsiveness continuously and without extra procedures
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Arterial Pressure Variation
Arterial blood pressure variation in GE patient monitoring
Systolic pressure variation (SPV) and pulse pressure variation (dPP) reect respiratory changes in arterial pressure during positive pressure ventilation.
SPV and dPP are calculated from the invasive arterial blood pressure using the following equations:
SPV [mmHg]= SBPmax - SBPmin
dPP [%]= (PPmax - PPmin) / [(PPmax + PPmin) / 2] * 100
SBPmax and SBPmin represent the maximum and minimum values of a systolic blood pressure over the measurement period, respectively.
SPV
PPmax and PPmin represent the maximum and minimum pulse pressures.
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min
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Values of SPV are represented in mmHg whereas dPP is a percentage. Both numbers are computed once every ve seconds. The measurement is continuous and trended in
the GE patient monitor. When the monitoring is started
it requires two respiratory cycles before the values are shown on the screen.
The arterial pressure variation algorithm analyzes the acquired invasive arterial blood pressure signal and, therefore, it does not require any further equipment besides the invasive blood pressure measurement kit.
Why monitor SPV/dPP?
One of the most common questions regarding hemodynamics has to do with the patient’s uid responsiveness: Will the patient’s stroke volume and cardiac output improve with uid resuscitation?
In hypovolemic conditions respiratory variations in
stroke volume are higher than normal responsive to uid therapy does not mean that the patient really needs uid.
Monitoring arterial pressure variation, i.e. SPV/dPP, helps to answer the question of uid responsiveness and can be used to guide uid expansion therapy parameters cannot, however, indicate what type of uid is the most suitable for the therapy.
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