General Electric MUSE RESTING ECG STATEMENT LIBRARY_SM_2002783-057_B Thermacare TC3146 Convective Warming Unit Service Manual

MUSE CV Information System Resting ECG Statement Library

(Software Ve rsion 005C)

Instructions

PN 2002783-057

$SABCO ND Aberrant conduction
$ACS ** Acute Cardiac Syndrome Criteria **

$AIS

$LBBBACS

$LBBBAIS

$LBBBAMI
$SAND and
$SANLERR1

$SANLERR2

$SANLERR3
$SCAPTUR Sinus/atrial capture

$SFUS Fusion complexes
$SIVR Idioventricular rhythm
$SOR or
$SRETC with retrograde conduction
$SVFIB Ventricular fibrillation
$SVTACH Ventricular tachycardia
$SWITH with
$SWQR Wide QRS rhythm
$TWLVW Leads V2, V3, V4 and V6 are interpolated
2ST with repolarization abnormality
AB Abnormal ECG

AB-VENT

ABER

ABER2
ABR Otherwise normal ECG

AC , possibly acute
ACCEL Accelerated
ACUMI *** ** ** ** * ACUTE MI ** ** ** **
ACUT Acute
AFB Left anterior fascicular block
AFIB Atrial fibrillation
AFL Atrial flutter
AFL-BL Atrial flutter with 2 to 1 block

AGSPAMI

AHE

AINJ Anterior injury pattern
AIOHAI
ALAD Abnormal left axis deviation

** ** With chest pain presence, consider
ACUTE ISCHMIA ** **

** ** With chest pain presence, consider
ACUTE MI if LBBB is new ** **

** ** Consider Acute Ischemia due to primary
ST-T change ** **

** ** Consider Acute MI due to primary ST-T
change ** **

*** Memory allocation failure, no ECG
interpretation possible

*** No QRS complexes found, no E CG analysis
possible

*** Less than 4 QRS complexes detected, no
interpretation possible

with intermittent aberrant ventricular
conduction

with premature ventricular or aberra nt l y
conducted complexes

with premature ventricular or aberra nt l y
conducted complexes

*** AGE AND GENDER SPECIFIC ECG
ANALYSIS ***

Acquisit i on hardware fault prevents reliable
analysis, carefully check ECG record before
interpreting

ST elevation consider anterior injury or acute
infarct

ALIHAI
ALINJ Anterolateral injury pattern

ALMI Anterolateral infarct
ALT

ALTWPW
AMI Anterior infarct

ANACP Manual comparison required for analog tracing
AND and
ANT Anterior leads
ANTLAT Anterolateral leads
ANT SEP Anteroseptal leads
APCK Electronic atrial pacemaker
ARAD Abnormal right axis deviation
ARAT (atrial rate
ARE-NO L are no longer
ARE-NOW are now

ARM

ASBINJ
ASINJ Anteroseptal injury pattern

ASM I Anterosepta l infarct
AT T wave abnormality, consider anterior ischemia
ATAC Atrial tachycardia
AU , age undetermined
AV-COND Suspect A-V conduction defect
AVDIS with A-V dissociation

AVPCK
AXIS QRS axis

BAE Biatrial en larg ement
BASIC Basic rhythm
BIFB *** Bifascicular block ***
BIFB1 (RBBB and left anterior fascicular block)
BIFB2 (RBBB and left posterior fascicular block)
BIGEM in a pattern of bigeminy
BIVH Biventricular hypertrophy
BLKED Blocked
BO Borderline
BOQTI Borderline QT interval
BORD-CRIT Borderline criteria for
BORDE Borderline ECG

CCWRT
CHB with com plete hea rt bloc k

CITED (cited on or before
CJP with a competing junctional pacemaker
COMP-H B Complete heart block
COMPAR When compared with ECG of

ST elevation consider anterolateral injury or
acute infarct

T wave abnormality, consider anterolateral
ischemia

with fusion or intermittent ventricular pre­excitation (WPW)

*** Suspect arm lead reversal, interpretation
assumes no reversal

Marked ST abnormality, possible anterior
subendocardial injury

AV sequential or dual chamber electronic
pacemaker

Counter clockwise rotation of the heart, may
invalidate criteria for ventricular hypertrophy

CONSEC Consecutive
CRI-FOR Criteria for
CRO Cannot rule out
CRS Coarse
CSEC , and consecutive

CUR-UND

CWRT
DEC-MI Questionable change in initial forces of

DEMOGR Warning: demographic data different
DICTATION Report dictated, transcription pending

DPCK

DTOFF
DXTRO Dextrocardia
EABRAD
EAR Unusual P axis, possible ectopic atrial rhythm

EARO Ectopic atrial rhythm
EATACH
EDP Electronic demand pacing

ESCBT with escape beat
EVO Serial changes of evolving
FAV with 1st degree A-V block
FLUT Atrial flutter
FREQ with frequent
HAS-CHG has changed
HAS-DE C has decreased
HAS-INC has increased
HAS-NO TCHG has not changed
HAS-RE P has replaced
HAV-CHG have changed
HAV-NOTCHG have not changed
HOWEVER however
HWV-IT however it
IDIO-R Idioventricular rhythm with AV block
IFLAT Inferolateral leads
IINJ Inferior injury pattern

IIOHAI
ILBBB Incomplete left bundle branch block
ILIHAI
ILINJ Inferolateral injury pattern
ILT
IMI Inferior infarct

INC-MI Increased evidence of infarction in
INDAX Indeterminate axis

Current undetermined rhythm precludes
rhythm comparison, needs review

Clockwise rotation of the heart, may invalidate
criteria for ventricular hypertrophy

Demand pacemaker; inter pret ation is bas ed on
intrinsic rhythm

Manual comparison required, data off line and
on volume

Unusual P axis, possible ectopic atrial
bradycardia

Unusual P axis, possible ectopic atrial
tachycardia

ST elevation consider inferior injury or acute
infarct

ST elevation consider inferolateral injury or
acute infarct

T wave abnormality, consider inferolateral
ischemia

INF Inferior leads
INFPOS Inferoposterior leads

INJONV
INVT-LOWT Inverted T waves have replaced flat T waves in

IPMI Inferior-posterior infarct
IRBBB Incomplete right bundle branch block
IRR Irregular
IRREG with undetermined rhythm irregularity

ISBINJ
IT T wave abnormality, consider inferior ischemia

IVCB Non-specific intra-ventricular conduction block
IVCD Non-specific intra-ventricular conduction delay
J-ESC with junctional escape
J-TACH Junctional tachycardia

JBRAD
JESC with junctional escape complexes
JR
JST Junctional ST depression, probably abnormal

JSTN Junctional ST depression, probably normal
JTACH
JUNBRAD Junctional bradycardia

JUNCT-R Junctional rhyt h m
LABRAD Left atrial bradycardia
LAD Leftward axis
LAD3 Left axis deviation
LAE Left atrial enlargement
LAR Left atrial rhythm
LARG Large
LAT Lateral leads
LATACH Left atrial tachycardia
LBBB Left bundle branch block
LESS-FLTT Fewer leads exhibit flat T waves in
LFREQ Less frequent
LHR Low heart rate, verify A-V conduction
LINJ Lateral injury pattern

LIOHAI
LMI Lateral infarct

LNGQT Prolonged QT
LOWT-INVT
LOWT-NOL Flat T waves no longer evident i n

LOWT-NOW Flat T waves now evident in
LOWV Low voltage QRS

LSBINJ
LT T wave abnormality, consider lateral ischemia

ST elevat ion, consider injury or variant
associated with LVH

Marked ST abnormality, possible inferior
subendocardial injury

Unusual P axis and short PR, probable
junctional bradycardia

Unusual P axis and short PR, probable
junctional rhythm

Unusual P axis and short PR, probable
junctional tachycardia

ST elevat ion consider lateral injury or acute
infarct

Flat T waves have replaced inverted T waves
in

Marked ST abnormality, possible lateral
subendocardial injury

Revision B MUSE CV information system
1 September 2002 2002783-057

LVH Voltage criteria for left ventricular hypertrophy
LVH2 Left ventricular hypertrophy

LVH3
MAFB (masked by fascicular block?)
MALT

MAT
MBZI with 2nd degr ee A- V block (Mob i tz I)

MBZII with 2nd degree A-V block (Mobitz II)
MFREQ More frequent

MILT
MINI-CRIT M inim a l criteria for
MISIZ

MIT

MLT
MOD Moderate

MORE-FLTT More leads exhibit flat T waves in
MRR
MSA R w ith marked sinus arr hythmia

MSBRA D Marked sinus bradycardia
MSTDAL

MSTDAS

MSTDIL
MULT-AT Multifocal atrial tachycardia

NEW , new
NML Normal ECG
NO-CHG No significant change was found

NO-SERCMP
NO-SERIAL No previous ECGs available

NOLONG is no longer
NOPF (no P-waves found)

NOPHONE
NOW is now

NQTACH Narrow QRS tachycardia
NSR Normal sinus rhythm
NST Nonspecific ST abnormality
NSTD Nonspecific ST depression
NSTE Nonspecific ST elevation

NSTFT
NSTLS Nonspecific T wave abnormality, improved in

NSTMR Nonspecific T wave abnormality, worse in
NSTNF

NSTNL

Moderate voltage criteria for LVH, may be
normal variant

Marked T wave abnormality, consider
anterolateral ischemia

Marked T wave abnormality, consider anterior
ischemia

Marked T-wave abnormality, consider
inferolateral ischemia

*** QRS contour suggests infarct size is
probably

Marked T wave abnormality, consider inferior
ischemia

Marked T wave abnormality, consider lateral
ischemia

Manual reading required due to inconsistent
morphologies

Marked ST abnormality, possible anterolateral
subendocardial injury

Marked ST abnormality, possible anteroseptal
subendocardial injury

Marked ST abnormality, possible inferolateral
subendocardial injury

Serial comparison not performed, all previous
tracings are of poor data quality

Manual comparison required, cannot contact
main system

Nonspecific T wave abnormality has replaced
inverted T waves in

Inverted T waves have replaced nonspecific T
wave abnormality in

Nonspecific T wave abnormality no longer
evident in

NSTNW Nonspecific T wave abnormality now evident in
NSTT Nonspecific ST and T wave abnormality
NT Nonspecific T wave abnormality
NWA Northwest axis
OCC with occasional
ODIG or digitalis effect
OLD , old
PAC Premature atrial complexes
PAUSE with sinus pause
PCARD Acute pericarditis
PCK Electronic ventricular pacemaker
PDIG , probably digitalis effect
PEC Premature ectopic complexes
PEDANL ** ** ** ** * Pediatric ECG Analysis * ** ** ** **
PFB Left posterior fascicular block
PJC Premature junctional complexes
PLV Prominent lateral voltage
PMDPV Prominent mid-precordial voltage,
PO Possible
PO-ATP Possible wandering atrial pacemaker

POOR-DAT
POS Posterior leads

POSTMI Posterior infarct
PPV Prominent posterior voltage

PR-SBRAD
PRESENT Present

PRINT PR interval
PRM-CON The premature contractions

PRV-UND
PSVC Premature supraventricular complexes

PULD Pulmonary disease pattern
PVC Premature ventricular complexes
PVCF Premature ventricular and fusion complexes
PXT , with posterior extension

QCERR

QESPMI
QIII Deep Q in lead III

QRS QRS
QRS-D UR QRS duration
QRS-VOL QRS voltage

QRST

QRSV
QRSW with QRS widening
QRSW-2ST
QT-LONG QT has lengthened

QT-SHRT QT has shortened
QUE-CHG Questionable change in
QUE-INICHG Questionable change in initial forces of

Poor data quality in current ECG precludes
serial comparison

Probable sinus bradycardia, verify A-V
conduction

Previous ECG has undetermined rhythm,
needs review

*** Poor data quality, interpretation may be
adversely affected

Increased R/S ratio in V1, consider early
transition or posterior infarct

Abnormal QRS-T angle, consider primary T
wave abnormality

Minimal voltage criteri a for L VH, may be normal
variant

with QRS widening and repolarization
abnormality

QUE-STCHG Questionable change in ST segment
QUE-TC HG Questionable change in T waves
QV6 Deep Q-wave in lead V6,
RABRAD Low right atrial bradycardia
RAD Rightward axis
RAD4 Right axis deviation
RAD5 Right superior axis deviation
RAE Right atrial enlargement
RAR Low right atrial rhythm
RAT-DEC Although rate has decreased
RAT-INC Although rate has increased
RATACH Low right atrial tachycardia
RBBB Right bundle branch block

RBBRVH
RECIP Reciprocal

REPOL Early repolarization
RHY Rhythm
RSAD Abnormal right superior axis deviation

RSR
RVE+ , plus right ventricular hypertrophy

RVH Right ventricular hypertrophy
RVH-2ST
RVR with rapid ventricular response
S1S2S3
SA-BLK with S-A block or transient A-V block

SAB with sinus arrest or transient A-V block
SABI with 2nd degree SA block (Mobitz I)
SABII with 2nd degree SA block (Mobitz II)
SAR with sinus arrhythmia
SAV with 2nd degree A-V block
SBRAD Sinus bradycardia
SEP Septal leads
SERCHG Serial changes of

SERYR1
SERYR2 ST elevation, consider early repolarization

SHFT-LFT Shifted left
SHFT-RGT Shifted right
SINJ Septal injury pattern
SMA Small
SMI Septal infarct
SNDQA , may be secondary to QRS abnormality
SNF Statement not found
SNGCH Significant changes have occurred
SPR with short PR
SRSRO RSR’ pattern in V1
SRTH Sinus rhythm

SSBINJ
ST& ST &

ST-(DEC) Non-specific change in ST segment in
ST-(INC) Non-specific change in ST segment in

Right bundle branch block -or- right ventricular
hypertrophy

RSR’ or QR pattern in V1 suggests right
ventricular conduction delay

Right ventricular hypertrophy with
repolarization abnormality

S1-S2-S3 pattern, consider pulmonary
disease, RVH, or normal variant

ST elevation, consider early repolarization,
pericarditis, or injury

Marked ST abnormality, possible septal
subendocardial injury

ST-DEPREP ST depression has replaced ST elevation in
ST-ELVPRS ST elevation now present in
ST-ELVREP ST elevation has replaced ST depression in
ST-LESDE P ST less depressed in
ST-LESELV ST less elevated in
ST-MORDEP ST more depressed
ST-MORELV ST more elevated in
ST-NOLDEP ST no longer depressed in
ST-NOLELV ST no longer elevated in
ST-NOWDEP ST now depressed in
STABAND ST abnormality and
STACH Sinus tachycardia

STDEP
STDIG ST abnormality, possible digitalis effect

STDPIN ST depression in
STELIN ST elevation in
SUNCNF (Unconfirmed)
SUP-TACH Supraventricular tachycardia
SVR with slow ventricular response
SVT Supraventricular tachycardia
T-DEC T wave amplitude has decreased in
T-INC T wave amplitude has increased in
T-INVMOR T wave inversion more evident in
T-INVNOL T wave inversion no longer evident in
T-INVNOW T wave inversion now evident in
T-LESINV T wave inversion les s evident in
T-WAVE T waves
TINVIN T-wave inversion in
TRIFB Trifascicular block
TVT with transient ventricular tachycardia
UR Undetermined rhythm
VAVB with variable A-V block
VENT-FUS with ventricular fusion
VENT-RAT Vent. rate
VENT-RTH Ventricular rhythm
VESC with ventricular escape complexes
VLAR Very large
VSMA Very small

VTACH
W2T1 with 2:1 A-V conduction

W3T1 with 3:1 A-V conduction
W4T1 with 4:1 A-V conduction
W5T1 with 5:1 A-V conduction
WEKH with Mobitz I (Wenckebach) block
WITH-D EM with a demand pacemaker
WITH-R ATDEC with rate decrease
WITH -RATINC with rate increase
WPW Wolff-Parkinson-White
WPWA Ventricular pre-excitation, WPW pattern type A
WPWB Ventricular pre-excitation, WPW pattern type B
WQTACH Wide QRS tachycardia
WSTR with strain pattern

ST depression, consider subendocardial injury
or digitalis effect

Ventricular tachycardia (ventricular or
supraventricular with aberration)

MUSE CV information system Revision B

2002783-057